Preprint established that higher concentrations of Galectin-9 led to more infection in terms of SARS-CoV-2 infected cells. The concept is that the Galectin-9 facilitates viral entry at a significantly higher rate than the control. In vitro studies also factored in and measured the expression of TMPRSS and ACE2 for a proper comparison. Reference to Sigamani et al study.

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All statements and expressions are the sole opinion of the editor and are subject to change without notice. The Company is not liable for any investment decisions by its readers or subscribers. It is strongly recommended that any purchase or sale decision be discussed with a financial advisor, or a broker-dealer, or a member of any financial regulatory bodies. The information contained herein has been provided as an information service only. The accuracy or completeness of the information is not warranted and is only as reliable as the sources from which it was obtained. Investors are cautioned that they may lose all or a portion of their investment in this or any other company. Information contained herein contains "forward looking statements" within the meaning of Section 27A of the Securities Act of 1933, as amended and Section 21E of the Securities and Exchange Act of 1934, as amended.

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Human Galectin-9 Potently Enhances SARS-CoV-2 Replication and Inflammation In Airway Epithelial Cells

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